Search results for "Microbial Mutation"

showing 5 items of 5 documents

Genome-Wide Estimation of the Spontaneous Mutation Rate of Human Adenovirus 5 by High-Fidelity Deep Sequencing

2016

Rates of spontaneous mutation determine the ability of viruses to evolve, infect new hosts, evade immunity and undergo drug resistance. Contrarily to RNA viruses, few mutation rate estimates have been obtained for DNA viruses, because their high replication fidelity implies that new mutations typically fall below the detection limits of Sanger and standard next-generation sequencing. Here, we have used a recently developed high-fidelity deep sequencing technique (Duplex Sequencing) to score spontaneous mutations in human adenovirus 5 under conditions of minimal selection. Based on >200 single-base spontaneous mutations detected throughout the entire viral genome, we infer an average mutatio…

0301 basic medicineAdenovirusesMutation rateGene Identification and AnalysisPathology and Laboratory MedicinePolymerase Chain ReactionMutation RateMedicine and Health Scienceslcsh:QH301-705.5GeneticsViral GenomicsInsertion MutationAdenovirus genomeMicrobial MutationHigh-Throughput Nucleotide SequencingGenomicsResistance mutation3. Good healthMedical MicrobiologyViral PathogensVirusesPathogensSequence AnalysisResearch Articlelcsh:Immunologic diseases. AllergySubstitution MutationImmunologyMicrobial GenomicsGenome ViralBiologyResearch and Analysis MethodsMicrobiologyDeep sequencingFrameshift mutation03 medical and health sciencesSequence Motif AnalysisVirologyGeneticsPoint MutationHumansMolecular Biology TechniquesSequencing TechniquesMicrobial PathogensMutation DetectionMolecular BiologySuppressor mutation030102 biochemistry & molecular biologyAdenoviruses HumanPoint mutationOrganismsBiology and Life SciencesVirology030104 developmental biologylcsh:Biology (General)MutationDynamic mutationParasitologyDNA viruseslcsh:RC581-607PLOS Pathogens
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Lamivudine/Adefovir Treatment Increases the Rate of Spontaneous Mutation of Hepatitis B Virus in Patients.

2016

The high levels of genetic diversity shown by hepatitis B virus (HBV) are commonly attributed to the low fidelity of its polymerase. However, the rate of spontaneous mutation of human HBV in vivo is currently unknown. Here, based on the evolutionary principle that the population frequency of lethal mutations equals the rate at which they are produced, we have estimated the mutation rate of HBV in vivo by scoring premature stop codons in 621 publicly available, full-length, molecular clone sequences derived from patients. This yielded an estimate of 8.7 × 10-5 spontaneous mutations per nucleotide per cell infection in untreated patients, which should be taken as an upper limit estimate becau…

0301 basic medicineMaleRNA virusesMutation ratelcsh:Medicinemedicine.disease_causeBiochemistryPolymerasesAdefovirFrameshift Mutationlcsh:SciencePathology and laboratory medicineeducation.field_of_studyMultidisciplinaryMicrobial MutationLamivudineMedical microbiologyResistance mutation3. Good healthLamivudineVirusesFemalePathogensSequence AnalysisImmunosuppressive Agentsmedicine.drugResearch ArticleHepatitis B virusSubstitution MutationPopulationOrganophosphonatesBiologyResearch and Analysis MethodsPolymorphism Single NucleotideMicrobiologyFrameshift mutation03 medical and health sciencesHepatitis B ChronicDrug Resistance ViralDNA-binding proteinsmedicineGeneticsHumanseducationMolecular Biology TechniquesSequencing TechniquesMolecular BiologyHepatitis B virusMedicine and health sciencesPoint mutationAdeninelcsh:RViral pathogensOrganismsBiology and Life SciencesProteinsVirologyMolecular biologyHepatitis virusesMicrobial pathogens030104 developmental biologyMutationlcsh:QCloningPLoS ONE
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Why are viral genomes so fragile? The bottleneck hypothesis

2021

If they undergo new mutations at each replication cycle, why are RNA viral genomes so fragile, with most mutations being either strongly deleterious or lethal? Here we provide theoretical and numerical evidence for the hypothesis that genetic fragility is partly an evolutionary response to the multiple population bottlenecks experienced by viral populations at various stages of their life cycles. Modelling within-host viral populations as multi-type branching processes, we show that mutational fragility lowers the rate at which Muller’s ratchet clicks and increases the survival probability through multiple bottlenecks. In the context of a susceptible-exposed-infectious-recovered epidemiolog…

Evolutionary GeneticsRNA virusesMutation rateEpidemiologyExtinct GenomesMedicine and Health SciencesBiology (General)Genetics0303 health sciencesEvolutionary epidemiologyEcologyMicrobial MutationGenomicsDeletion MutationComputational Theory and MathematicsViral genomesGenetic EpidemiologyModeling and SimulationViral evolutionPopulation bottlenecksVirusesRNA ViralResearch ArticleQH301-705.5Genomics[SDV.CAN]Life Sciences [q-bio]/CancerContext (language use)Genome ViralBiologyMicrobiologyGenomic InstabilityViral EvolutionBottleneckEvolution Molecular03 medical and health sciencesCellular and Molecular NeuroscienceSurvival probabilityVirologyGeneticsFragilityMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyEvolutionary BiologyModels Genetic030306 microbiologyOrganismsComputational BiologyBiology and Life SciencesRNAVirus evolutionOrganismal EvolutionGenetic architecture[MATH.MATH-PR]Mathematics [math]/Probability [math.PR]Population bottleneckViral replicationMutationMicrobial Evolution
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From molecular genetics to phylodynamics: evolutionary relevance of mutation rates across viruses.

2012

Although evolution is a multifactorial process, theory posits that the speed of molecular evolution should be directly determined by the rate at which spontaneous mutations appear. To what extent these two biochemical and population-scale processes are related in nature, however, is largely unknown. Viruses are an ideal system for addressing this question because their evolution is fast enough to be observed in real time, and experimentally-determined mutation rates are abundant. This article provides statistically supported evidence that the mutation rate determines molecular evolution across all types of viruses. Properties of the viral genome such as its size and chemical composition are…

Evolutionary Geneticslcsh:Immunologic diseases. AllergyMutation rateGenome evolutionImmunologyGenome ViralBiologyGenomeMicrobiologyEvolution Molecular03 medical and health sciencesMutation RateMolecular evolutionPhylogeneticsVirologyGeneticsMolecular BiologyBiologylcsh:QH301-705.5Phylogeny030304 developmental biologyGenetics0303 health sciencesEvolutionary BiologyModels Genetic030306 microbiologyMutagenesisMicrobial MutationBiological EvolutionViral phylodynamicslcsh:Biology (General)Viral evolutionVirusesParasitologylcsh:RC581-607Population GeneticsResearch ArticlePLoS Pathogens
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Large-scale analysis of SARS-CoV-2 spike-glycoprotein mutants demonstrates the need for continuous screening of virus isolates

2021

Due to the widespread of the COVID-19 pandemic, the SARS-CoV-2 genome is evolving in diverse human populations. Several studies already reported different strains and an increase in the mutation rate. Particularly, mutations in SARS-CoV-2 spike-glycoprotein are of great interest as it mediates infection in human and recently approved mRNA vaccines are designed to induce immune responses against it. We analyzed 1,036,030 SARS-CoV-2 genome assemblies and 30,806 NGS datasets from GISAID and European Nucleotide Archive (ENA) focusing on non-synonymous mutations in the spike protein. Only around 2.5% of the samples contained the wild-type spike protein with no variation from the reference. Among…

RNA virusesMutation rateCoronavirusesEpidemiologyMolecular biologyT-LymphocytesMutantGene Identification and Analysismedicine.disease_causeGenomeWhite Blood CellsDatabase and Informatics MethodsSequencing techniquesMutation RateAnimal CellsDNA sequencingPathology and laboratory medicineGeneticsMutationMultidisciplinaryT CellsMicrobial MutationQRHigh-Throughput Nucleotide SequencingGenomicsMedical microbiologyVirusesSpike Glycoprotein CoronavirusMedicineSARS CoV 2PathogensCellular TypesTranscriptome AnalysisSequence AnalysisResearch ArticleNext-Generation SequencingSARS coronavirusBioinformaticsImmune CellsScienceImmunologyProtein domainSequence alignmentGenomicsGenome ViralBiologyMicrobiologyAntibodiesDNA sequencingProtein DomainsGeneticsmedicineHumansMutation DetectionPandemicsMedicine and health sciencesBlood CellsBiology and life sciencesSARS-CoV-2OrganismsViral pathogensComputational BiologyCOVID-19Cell BiologyGenome AnalysisMicrobial pathogensResearch and analysis methodsMolecular biology techniquesMutationSequence AlignmentPLOS ONE
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